{"id":1360,"date":"2026-04-06T15:00:00","date_gmt":"2026-04-06T15:00:00","guid":{"rendered":"https:\/\/lotilabs.com\/resources\/?p=1360"},"modified":"2026-09-07T18:50:28","modified_gmt":"2026-09-07T18:50:28","slug":"semax-peptide-neurotrophic-research-guide","status":"publish","type":"post","link":"https:\/\/lotilabs.com\/resources\/semax-peptide-neurotrophic-research-guide\/","title":{"rendered":"Semax Peptide: Neurotrophic Research, Cognitive Studies &#038; Laboratory Applications"},"content":{"rendered":"<div class=\"ez-toc-v2_0_83 counter-hierarchy ez-toc-counter ez-toc-light-blue ez-toc-container-direction\" id=\"ez-toc-container\">\n<div class=\"ez-toc-title-container\">\n<p class=\"ez-toc-title\" style=\"cursor:inherit\">Table of Contents<\/p>\n<span class=\"ez-toc-title-toggle\"><a aria-label=\"Toggle Table of Content\" class=\"ez-toc-pull-right ez-toc-btn ez-toc-btn-xs ez-toc-btn-default ez-toc-toggle\" href=\"#\"><span class=\"ez-toc-js-icon-con\"><span class=\"\"><span class=\"eztoc-hide\" style=\"display:none;\">Toggle<\/span><span class=\"ez-toc-icon-toggle-span\"><svg class=\"list-377408\" fill=\"none\" height=\"20px\" style=\"fill: #999;color:#999\" viewbox=\"0 0 24 24\" width=\"20px\" xmlns=\"http:\/\/www.w3.org\/2000\/svg\"><path d=\"M6 6H4v2h2V6zm14 0H8v2h12V6zM4 11h2v2H4v-2zm16 0H8v2h12v-2zM4 16h2v2H4v-2zm16 0H8v2h12v-2z\" fill=\"currentColor\"><\/path><\/svg><svg baseprofile=\"tiny\" class=\"arrow-unsorted-368013\" height=\"10px\" style=\"fill: #999;color:#999\" version=\"1.2\" viewbox=\"0 0 24 24\" width=\"10px\" xmlns=\"http:\/\/www.w3.org\/2000\/svg\"><path d=\"M18.2 9.3l-6.2-6.3-6.2 6.3c-.2.2-.3.4-.3.7s.1.5.3.7c.2.2.4.3.7.3h11c.3 0 .5-.1.7-.3.2-.2.3-.5.3-.7s-.1-.5-.3-.7zM5.8 14.7l6.2 6.3 6.2-6.3c.2-.2.3-.5.3-.7s-.1-.5-.3-.7c-.2-.2-.4-.3-.7-.3h-11c-.3 0-.5.1-.7.3-.2.2-.3.5-.3.7s.1.5.3.7z\"><\/path><\/svg><\/span><\/span><\/span><\/a><\/span><\/div>\n<nav><ul class=\"ez-toc-list ez-toc-list-level-1\"><li class=\"ez-toc-page-1 ez-toc-heading-level-2\"><a class=\"ez-toc-link ez-toc-heading-2\" href=\"https:\/\/lotilabs.com\/resources\/semax-peptide-neurotrophic-research-guide\/#What_Is_Semax\">What Is Semax?<\/a><\/li><li class=\"ez-toc-page-1 ez-toc-heading-level-2\"><a class=\"ez-toc-link ez-toc-heading-3\" href=\"https:\/\/lotilabs.com\/resources\/semax-peptide-neurotrophic-research-guide\/#Development_Story\">Development Story<\/a><\/li><li class=\"ez-toc-page-1 ez-toc-heading-level-2\"><a class=\"ez-toc-link ez-toc-heading-4\" href=\"https:\/\/lotilabs.com\/resources\/semax-peptide-neurotrophic-research-guide\/#Chemistry_and_Structure\">Chemistry and Structure<\/a><\/li><li class=\"ez-toc-page-1 ez-toc-heading-level-2\"><a class=\"ez-toc-link ez-toc-heading-5\" href=\"https:\/\/lotilabs.com\/resources\/semax-peptide-neurotrophic-research-guide\/#Mechanism_of_Action_BDNF_Is_the_Headline_But_Not_the_Whole_Story\">Mechanism of Action: BDNF Is the Headline, But Not the Whole Story<\/a><\/li><li class=\"ez-toc-page-1 ez-toc-heading-level-2\"><a class=\"ez-toc-link ez-toc-heading-6\" href=\"https:\/\/lotilabs.com\/resources\/semax-peptide-neurotrophic-research-guide\/#Preclinical_Findings_Stroke_Cognition_and_Gene_Expression\">Preclinical Findings: Stroke, Cognition, and Gene Expression<\/a><\/li><li class=\"ez-toc-page-1 ez-toc-heading-level-2\"><a class=\"ez-toc-link ez-toc-heading-7\" href=\"https:\/\/lotilabs.com\/resources\/semax-peptide-neurotrophic-research-guide\/#Semax_vs_Selank_Same_Lab_Different_Compounds\">Semax vs. Selank: Same Lab, Different Compounds<\/a><\/li><li class=\"ez-toc-page-1 ez-toc-heading-level-2\"><a class=\"ez-toc-link ez-toc-heading-8\" href=\"https:\/\/lotilabs.com\/resources\/semax-peptide-neurotrophic-research-guide\/#Safety_Profile\">Safety Profile<\/a><\/li><li class=\"ez-toc-page-1 ez-toc-heading-level-2\"><a class=\"ez-toc-link ez-toc-heading-9\" href=\"https:\/\/lotilabs.com\/resources\/semax-peptide-neurotrophic-research-guide\/#Regulatory_Status\">Regulatory Status<\/a><\/li><li class=\"ez-toc-page-1 ez-toc-heading-level-2\"><a class=\"ez-toc-link ez-toc-heading-10\" href=\"https:\/\/lotilabs.com\/resources\/semax-peptide-neurotrophic-research-guide\/#Research_Availability\">Research Availability<\/a><\/li><li class=\"ez-toc-page-1 ez-toc-heading-level-2\"><a class=\"ez-toc-link ez-toc-heading-11\" href=\"https:\/\/lotilabs.com\/resources\/semax-peptide-neurotrophic-research-guide\/#Conclusion\">Conclusion<\/a><\/li><li class=\"ez-toc-page-1 ez-toc-heading-level-2\"><a class=\"ez-toc-link ez-toc-heading-12\" href=\"https:\/\/lotilabs.com\/resources\/semax-peptide-neurotrophic-research-guide\/#References\">References<\/a><\/li><\/ul><\/nav><\/div>\n\n<p><em>For laboratory and research use only. Not for human consumption.<\/em><\/p>\n<p>That\u2019s all it took. Within hours, BDNF mRNA in the hippocampus tripled. TrkB phosphorylation climbed 1.6-fold. A single administration.<\/p>\n<p>Data like that is why <a href=\"https:\/\/lotilabs.com\/product\/semax-5mg\/\">Semax<\/a> went from a niche Russian neuropeptide to one of the most studied nootropic compounds in neuroscience research. It\u2019s a seven-amino-acid synthetic peptide (Met-Glu-His-Phe-Pro-Gly-Pro) derived from the ACTH(4\u201310) fragment. The Pro-Gly-Pro tail bolted onto the C-terminus makes it resistant to enzymatic chewing \u2014 and, it turns out, carries independent pharmacological activity of its own. Three-plus decades in research. Registered in Russia since 1996. That list sounds implausible for one peptide. Then you read the published papers and realize every pathway has data behind it.<\/p>\n<h2><span class=\"ez-toc-section\" id=\"What_Is_Semax\"><\/span><span class=\"ez-toc-section\" id=\"What_Is_Semax\"><\/span>What Is Semax?<span class=\"ez-toc-section-end\"><\/span><span class=\"ez-toc-section-end\"><\/span><\/h2>\n<p>Seven amino acids. MEHFPGP. A synthetic analogue of the ACTH(4\u201310) fragment \u2014 that\u2019s the stretch of adrenocorticotropic hormone between residues 4 and 10 that carries the CNS activity. The full ACTH molecule stimulates cortisol release. Semax doesn\u2019t. Zero hormonal activity. All the brain effects, none of the adrenal stimulation.<\/p>\n<ul>\n<li><strong>Sequence:<\/strong> Met-Glu-His-Phe-Pro-Gly-Pro (MEHFPGP)<\/li>\n<li><strong>Classification:<\/strong> Synthetic ACTH(4\u201310) analogue with PGP extension<\/li>\n<li><strong>Molecular Weight:<\/strong> ~813.97 g\/mol<\/li>\n<li><strong>Route (preclinical):<\/strong> Predominantly intranasal<\/li>\n<li><strong>Status:<\/strong> Investigational research compound; not FDA-approved<\/li>\n<\/ul>\n<p>The PGP tail isn\u2019t just armor against peptidases. Two pharmacological programs in one peptide.<\/p>\n<h2><span class=\"ez-toc-section\" id=\"Development_Story\"><\/span><span class=\"ez-toc-section\" id=\"Development_Story\"><\/span>Development Story<span class=\"ez-toc-section-end\"><\/span><span class=\"ez-toc-section-end\"><\/span><\/h2>\n<p>1980s. Nikolay Myasoedov\u2019s lab at the Institute of Molecular Genetics, Russian Academy of Sciences. Same institute that produced <a href=\"https:\/\/lotilabs.com\/product\/selank-10mg\/\">Selank<\/a>. The goal: take the ACTH(4\u201310) fragment \u2014 already known to have CNS effects \u2014 and make it last long enough to be useful. Intranasal delivery. No hormonal baggage.<\/p>\n<p>They succeeded. It\u2019s still available there and in Ukraine as a mist applicator. Three decades of published research, mostly in Russian-language journals \u2014 though international work is picking up as transcriptomic and proteomic tools make Semax\u2019s multi-pathway effects easier to characterize at a molecular level.<\/p>\n<p>No FDA approval. No EMA approval. In the West, Semax remains strictly a research compound. That hasn\u2019t slowed the accumulation of preclinical data.<\/p>\n<h2><span class=\"ez-toc-section\" id=\"Chemistry_and_Structure\"><\/span><span class=\"ez-toc-section\" id=\"Chemistry_and_Structure\"><\/span>Chemistry and Structure<span class=\"ez-toc-section-end\"><\/span><span class=\"ez-toc-section-end\"><\/span><\/h2>\n<p>Start with ACTH\u2019s core pharmacophore \u2014 the His-Phe sequence at positions 6-7 that drives melanocortin-related CNS activity. Semax modifies the surrounding residues (swapping Arg-Trp for Pro-Gly-Pro) so you keep the brain activity but eliminate adrenal stimulation. The Met-Glu N-terminus and the PGP C-terminus are what make Semax a distinct compound rather than just an ACTH fragment.<\/p>\n<p>That PGP tail warrants attention.<\/p>\n<h2><span class=\"ez-toc-section\" id=\"Mechanism_of_Action_BDNF_Is_the_Headline_But_Not_the_Whole_Story\"><\/span><span class=\"ez-toc-section\" id=\"Mechanism_of_Action_BDNF_Is_the_Headline_But_Not_the_Whole_Story\"><\/span>Mechanism of Action: BDNF Is the Headline, But Not the Whole Story<span class=\"ez-toc-section-end\"><\/span><span class=\"ez-toc-section-end\"><\/span><\/h2>\n<p>Semax\u2019s nootropic effects run through multiple pathways. BDNF\/TrkB gets the most attention. Rightfully \u2014 the numbers are striking. But there\u2019s more happening underneath.<\/p>\n<h3>BDNF and TrkB: The Core Pathway<\/h3>\n<p>The landmark data comes from a Brain Research paper (PMID: 16996037). Hippocampal effects:<\/p>\n\n<p>Hours. That\u2019s not a marginal effect \u2014 that\u2019s a robust neurotrophic signal.<\/p>\n<p>Binding studies went further: tritium-labeled Semax binds specific sites in rat basal forebrain with a K<sub>D<\/sub> of 2.4 \u00b1 1.0 nM and B<sub>MAX<\/sub> of 33.5 \u00b1 7.9 fmol\/mg protein. Calcium-dependent. Reversible. That\u2019s receptor-mediated binding, not nonspecific membrane interaction. And the BDNF response was region-specific \u2014 basal forebrain yes, cerebellum no. Targeted, not diffuse.<\/p>\n<h3>NGF and Neurotrophin-3<\/h3>\n<p>BDNF isn\u2019t alone. In permanent middle cerebral artery occlusion (pMCAO) models, Semax upregulated NGF, NT-3, and their high-affinity receptors (TrkA, TrkB, TrkC). Timing and magnitude varied by brain region and how long since the ischemic event (PMC11498467). That broad neurotrophin-activating profile is unusual \u2014 most compounds in this space hit one pathway, maybe two. Semax hits the full set.<\/p>\n<h3>Dopamine and Serotonin<\/h3>\n<p>Eremin et al. Serotonergic modulation runs alongside.<\/p>\n<h2><span class=\"ez-toc-section\" id=\"Preclinical_Findings_Stroke_Cognition_and_Gene_Expression\"><\/span><span class=\"ez-toc-section\" id=\"Preclinical_Findings_Stroke_Cognition_and_Gene_Expression\"><\/span>Preclinical Findings: Stroke, Cognition, and Gene Expression<span class=\"ez-toc-section-end\"><\/span><span class=\"ez-toc-section-end\"><\/span><\/h2>\n\n<p>This is some of the most compelling Semax data. Genome-wide expression profiling (Illumina RatRef-12 BeadChip) on cortical tissue from pMCAO rodents (PMC3987924):<\/p>\n<p>Immunoglobulin genes were the most heavily upregulated group \u2014 several showing the highest expression amplitude of any transcript affected. Vascular system and intracellular calcium genes also moved.<\/p>\n\n<p>Consistent results across multiple rodent behavioral paradigms. Attention sharpens.<\/p>\n<p>Resting-state fMRI work adds anatomical context: Semax produces measurable changes in default mode network architecture, specifically the medial frontal cortex subcomponent. You can see the compound\u2019s effects on functional brain connectivity, not just isolated neurotransmitter measures.<\/p>\n<h3>Transcriptomic Depth<\/h3>\n<p>In the intact rat hippocampus (no ischemia, no pathology), Semax modulates genes involved in intracellular signaling: MAPK10, APP (amyloid precursor protein), fibronectin 1. This tells you something important \u2014 Semax isn\u2019t only active under pathological conditions.<\/p>\n<p>For labs studying neuropeptide signaling in cognition, <a href=\"https:\/\/lotilabs.com\/resources\/delta-sleep-inducing-peptide-dsip-benefits-uses-and-dosage\/\">DSIP<\/a> and <a href=\"https:\/\/lotilabs.com\/resources\/the-benefits-of-pinealon-peptide-for-brain-and-longevity-research\/\">Pinealon<\/a> are relevant comparisons.<\/p>\n<h2><span class=\"ez-toc-section\" id=\"Semax_vs_Selank_Same_Lab_Different_Compounds\"><\/span><span class=\"ez-toc-section\" id=\"Semax_vs_Selank_Same_Lab_Different_Compounds\"><\/span>Semax vs. Selank: Same Lab, Different Compounds<span class=\"ez-toc-section-end\"><\/span><span class=\"ez-toc-section-end\"><\/span><\/h2>\n<p>Both heptapeptides. Both from Myasoedov\u2019s institute. Both share the PGP tail. But they do fundamentally different things.<\/p>\n<table>\n<thead>\n<tr>\n<th>Parameter<\/th>\n<th>Semax<\/th>\n<th>Selank<\/th>\n<\/tr>\n<\/thead>\n<tbody>\n<tr>\n<td><strong>Parent<\/strong><\/td>\n<td>ACTH(4\u201310)<\/td>\n<td>Tuftsin<\/td>\n<\/tr>\n<tr>\n<td><strong>Sequence<\/strong><\/td>\n<td>MEHFPGP<\/td>\n<td>TKPRPGP<\/td>\n<\/tr>\n<tr>\n<td><strong>Primary Profile<\/strong><\/td>\n\n<td>Anxiolytic, immunomodulatory<\/td>\n<\/tr>\n<tr>\n<td><strong>Core Mechanism<\/strong><\/td>\n<td>BDNF\/NGF upregulation, DA\/5-HT modulation<\/td>\n<td>GABA<sub>A<\/sub> modulation, enkephalinase inhibition<\/td>\n<\/tr>\n<tr>\n<td><strong>BDNF Effect<\/strong><\/td>\n<td>Primary: 1.4\u00d7 protein, 3\u00d7 mRNA<\/td>\n<td>Secondary: moderate, state-dependent<\/td>\n<\/tr>\n\n<tr>\n<td><strong>Anxiolytic<\/strong><\/td>\n\n<td>Strong \u2014 benzo-equivalent without sedation<\/td>\n<\/tr>\n<tr>\n<td><strong>Russia Approval<\/strong><\/td>\n<td>1996<\/td>\n<td>2009<\/td>\n<\/tr>\n<tr>\n<td><strong>Gene Expression<\/strong><\/td>\n\n<td>45 neurotransmission genes in frontal cortex<\/td>\n<\/tr>\n<\/tbody>\n<\/table>\n<p>Quick version: Semax is the cognition compound. Both touch neurotrophins. But their primary research domains don\u2019t overlap much.<\/p>\n<h2><span class=\"ez-toc-section\" id=\"Safety_Profile\"><\/span><span class=\"ez-toc-section\" id=\"Safety_Profile\"><\/span>Safety Profile<span class=\"ez-toc-section-end\"><\/span><span class=\"ez-toc-section-end\"><\/span><\/h2>\n<p>Across every published preclinical study \u2014 clean. No hormonal effects despite the ACTH heritage. Semax does not trigger corticosterone or cortisol release.<\/p>\n<p>Protocol design notes:<\/p>\n<ul>\n<li>The ACTH parentage is misleading \u2014 hormonal activity was specifically engineered out. No adrenal stimulation<\/li>\n<li>Binding is calcium-dependent. Control for that in your in vitro work or you\u2019ll get artifacts<\/li>\n<li>BDNF response is region-specific: basal forebrain and hippocampus respond strongly, cerebellum doesn\u2019t budge. Sample your tissues accordingly<\/li>\n<li>PGP tail makes Semax resistant to aminopeptidases. That changes your PK modeling assumptions<\/li>\n<\/ul>\n<p>Procurement basics: demand third-party COAs, HPLC purity data, and mass spec confirmation. Peptide aggregation and impurity risks are real with any research peptide.<\/p>\n<h2><span class=\"ez-toc-section\" id=\"Regulatory_Status\"><\/span><span class=\"ez-toc-section\" id=\"Regulatory_Status\"><\/span>Regulatory Status<span class=\"ez-toc-section-end\"><\/span><span class=\"ez-toc-section-end\"><\/span><\/h2>\n<p><strong>Russia\/Ukraine:<\/strong> Registered. One of the few peptides with actual regulatory approval for CNS indications anywhere.<\/p>\n<p><strong>United States:<\/strong> Not FDA-approved for any indication. Research compound only. Not a controlled substance.<\/p>\n<p><strong>EU\/International:<\/strong> Not EMA-approved. Classified as investigational.<\/p>\n<h2><span class=\"ez-toc-section\" id=\"Research_Availability\"><\/span><span class=\"ez-toc-section\" id=\"Research_Availability\"><\/span>Research Availability<span class=\"ez-toc-section-end\"><\/span><span class=\"ez-toc-section-end\"><\/span><\/h2>\n<p><a href=\"https:\/\/lotilabs.com\/product\/semax-5mg\/\">Semax 5mg<\/a> from Loti Labs \u2014 third-party verified purity, suitable for in vitro and in vivo work. Researchers doing comparative neuropeptide pharmacology should also consider <a href=\"https:\/\/lotilabs.com\/product\/selank-10mg\/\">Selank 10mg<\/a> for parallel or combination studies.<\/p>\n<h2><span class=\"ez-toc-section\" id=\"Conclusion\"><\/span><span class=\"ez-toc-section\" id=\"Conclusion\"><\/span>Conclusion<span class=\"ez-toc-section-end\"><\/span><span class=\"ez-toc-section-end\"><\/span><\/h2>\n<p>Three decades of data and counting. Region-specific neurotrophic effects verified by binding studies with nanomolar affinity.<\/p>\n<p>Semax isn\u2019t a simple nootropic. The receptor it binds to hasn\u2019t been definitively identified yet (K<sub>D<\/sub> = 2.4 nM, but the molecular target remains unknown). Neurodegenerative disease models are largely unexplored. And combination protocols with Selank and other neuropeptides are barely off the ground.<\/p>\n<p>There\u2019s a lot of open territory here. For neuroscience labs with the right tools, Semax is one of the more interesting compounds to put under the microscope.<\/p>\n<p><em>For laboratory and research use only. Not for human consumption.<\/em><\/p>\n<h2><span class=\"ez-toc-section\" id=\"References\"><\/span><span class=\"ez-toc-section\" id=\"References\"><\/span>References<span class=\"ez-toc-section-end\"><\/span><span class=\"ez-toc-section-end\"><\/span><\/h2>\n<ol>\n<li>Dolotov OV, et al. <em>Brain Res.<\/em> 2006;1117(1):54-60. PMID: 16996037<\/li>\n<li>Dergunova LV, et al. <em>BMC Genomics.<\/em> 2014;15:228. PMC3987924<\/li>\n<li>Filippenkov IB, et al. Novel Insights into the Protective Properties of ACTH(4-7)PGP (Semax) Peptide at the Transcriptome Level Following Cerebral Ischaemia\u2013Reperfusion in Rats. <em>Genes.<\/em> 2020. PMC7555804<\/li>\n<li>Dolotov OV, et al. <em>J Neurochem.<\/em> 2006;97 Suppl 1:82-86.<\/li>\n<li>Eremin KO, et al. Semax, An ACTH(4-10) Analogue with Nootropic Properties, Activates Dopaminergic and Serotoninergic Brain Systems in Rodents. <em>Neurochem Res.<\/em> 2005;30:1493-1500.<\/li>\n<li>Dolotov OV, et al. The heptapeptide SEMAX stimulates BDNF expression in different areas of the rat brain in vivo. <em>Doklady Biol Sci.<\/em> 2004;399:478-481.<\/li>\n<li>Filippenkov IB, et al. Brain Protein Expression Profile Confirms the Protective Effect of the ACTH(4\u20137)PGP Peptide (Semax) in a Rat Model of Cerebral Ischemia\u2013Reperfusion. <em>Int J Mol Sci.<\/em> 2021.<\/li>\n<li>Agapova TY, et al. Semax and Pro-Gly-Pro Activate the Transcription of Neurotrophins and Their Receptor Genes after Cerebral Ischemia. <em>Cell Mol Neurobiol.<\/em> 2009. PMC11498467<\/li>\n<li>Ashmarin IP, et al. A nootropic adrenocorticotropin analog 4-10-semax (15 years experience in its design and study). <em>Zh Vyssh Nerv Deiat.<\/em> 1997;47(2):420-430.<\/li>\n<\/ol>\n","protected":false},"excerpt":{"rendered":"<p>Table of Contents Toggle What Is Semax?Development StoryChemistry and StructureMechanism of Action: BDNF Is the Headline, But Not the Whole StoryPreclinical Findings: Stroke, Cognition, and Gene ExpressionSemax vs. Selank: Same Lab, Different CompoundsSafety ProfileRegulatory StatusResearch AvailabilityConclusionReferences For laboratory and research use only. Not for human consumption. That\u2019s all it took. Within hours, BDNF mRNA in [&#8230;]\n","protected":false},"author":1,"featured_media":1385,"comment_status":"closed","ping_status":"closed","sticky":false,"template":"","format":"standard","meta":{"footnotes":""},"categories":[5],"tags":[],"class_list":["post-1360","post","type-post","status-publish","format-standard","has-post-thumbnail","hentry","category-peptides"],"_links":{"self":[{"href":"https:\/\/lotilabs.com\/resources\/wp-json\/wp\/v2\/posts\/1360","targetHints":{"allow":["GET"]}}],"collection":[{"href":"https:\/\/lotilabs.com\/resources\/wp-json\/wp\/v2\/posts"}],"about":[{"href":"https:\/\/lotilabs.com\/resources\/wp-json\/wp\/v2\/types\/post"}],"author":[{"embeddable":true,"href":"https:\/\/lotilabs.com\/resources\/wp-json\/wp\/v2\/users\/1"}],"replies":[{"embeddable":true,"href":"https:\/\/lotilabs.com\/resources\/wp-json\/wp\/v2\/comments?post=1360"}],"version-history":[{"count":0,"href":"https:\/\/lotilabs.com\/resources\/wp-json\/wp\/v2\/posts\/1360\/revisions"}],"wp:featuredmedia":[{"embeddable":true,"href":"https:\/\/lotilabs.com\/resources\/wp-json\/wp\/v2\/media\/1385"}],"wp:attachment":[{"href":"https:\/\/lotilabs.com\/resources\/wp-json\/wp\/v2\/media?parent=1360"}],"wp:term":[{"taxonomy":"category","embeddable":true,"href":"https:\/\/lotilabs.com\/resources\/wp-json\/wp\/v2\/categories?post=1360"},{"taxonomy":"post_tag","embeddable":true,"href":"https:\/\/lotilabs.com\/resources\/wp-json\/wp\/v2\/tags?post=1360"}],"curies":[{"name":"wp","href":"https:\/\/api.w.org\/{rel}","templated":true}]}}